Environmental Pollution exposure unlock the pathogenesis of Lung Cancer: An Updates

Abstract

Air pollution and lung cancer are global issues. This study examines how PM2.5 and PAHs cause and accelerate lung cancer. Due to environmental and occupational contaminants, lung cancer is common among nonsmokers. Global and national regulations are needed to govern air pollution emissions and design PPE to avoid lung injuries, considering physiological systems. The analysis highlights the need for effective preventative measures and laws to reduce the influence of air pollution on lung cancer rates. It provides extensive insights into the link between cancer-causing substances and lung cancer. The work discusses genetic predispositions, environmental exposures, and occupational risks that cause lung cancer, focusing on tobacco smoke and secondary exposure. Lung cancer risk is rising due to air pollution, prompting preventative and regulatory measures. Carcinogenic pollutants cause multiple cancers. Benzene, formaldehyde, arsenic, PVC, PAHs, nitrosamines, and other carcinogenic pollutants are discussed in this work. To reduce cancer risk, regulations, public health initiatives, and personal preventive measures should be implemented. They are controlling cancer-causing contaminants. Legislation, personal safety, public awareness, and education are included. Education, public-private partnerships, community participation, and media exposure are also encouraged—the significance of regular updates and monitoring in informing the public about environmental health advances. Individuals, communities, and governments are working to reduce cancer-causing toxins and improve the environment.